Read up on how the body regulates copper levels and why Selenium gets
depleted.
Silver is handled the same way, but there is no need for silver as there
is for small amounts of copper...and... it's hard to AVOID copper.
Has to do with metallo transport proteins and how they work. [Dunno where
that ties in with liver function, but prolly does]
It seems reasonable that if a lot of copper is around, Selenium, being a
bit rare and hard to find, will be low and you'll be susceptible to silver
retention as the bodies #2 [or #10] priority for elimination...copper being
toxic if not properly regulated [...and likely zinc, I think] and silver
being non toxic, NOT being a micro nutrient trace element that *needs*
regulating. [Or Else]
Also seems reasonable that if you NEED high levels of silver for a while,
that supplementing copper etc would increase them.
Load up the bus with folx that MUST leave town or the town riots, the
tourists get another day to hang out....no room on the bus.
Ode
At 10:58 AM 6/3/2009 -0500, you wrote:
From âThe Ultimate Colloidal Silver Manual":
âMany colloidal silver users claim that argyria is caused when the liver
and kidneys become so overwhelmed with excessive silver intake, that they
cannot process it from the body as quickly as it is being taken into the
body. This assertion makes sense to us, though we have yet to see the
medical evidence for it. Many of these users also assert that taking 200
mcg. of selenium and 400 IU of vitamin E daily will help protect the liver
from potentially toxic effects of excessive silver intake. This contention
is apparently based on animal studies conducted in the late 1960âs and
early 1970âs, which apparently demonstrated that when rats were fed
extremely high levels of silver in water, relative to their body weight
(140 mg/kg/day), they developed a deficiency of selenium which in turn
inhibited the synthesis of the seleno-enzyme glutathione peroxidase (which
is crucial to proper liver function). This apparently caused liver
toxicity. Interestingly, when the researchers added small amounts of
selenium and vitamin E to the ratâs daily diets, the liver damage was
completely eliminated, even though the excessively high daily levels of
silver water were still being given to the rats. The daily dosage of
selenium and vitamin E given to the rats were later extrapolated to human
usage as being 200 mcg. of selenium and 400 IU of vitamin E. But while
this is intriguing information, and certainly worthy of further study, we
must warn that to our knowledge there is absolutely no confirming medical
research to demonstrate that this will work in humans in the same way it
apparently worked in rats."
There is an anecdote on the internet of a claimed reversal of argyria but
to my knowledge no one else has claimed success with it:
âAbout two months ago, I was contacted by an individual with an
amazing story: This individual utilized 32 ounces of silver chloride
daily for 2.5 years and cured a very late stage case of neurosyphilis.
individual to become argyric. The silver chloride was produced in ten
minute batches using the 3 nine's method of production with no controls.
I have since spent quite some time on the phone with this individual.
The second part of this story: By ingesting large amounts of a high
concentration silver salt, the individual acquired argyria. It was likely
a bit more severe than Stan Jones, but certainly not an aggravated
condition like Rosemary Jacobs' agryria. It was bad enough to turn heads
in a grocery store. However, this individual didn't stop with eliminating
his lethal condition. He also reversed the argyria.
Many have long discussed the possibility of using Vitamin E, Selenium, and
other supplements with a cleansing program to reverse the condition,
however, we've never seen someone actually accomplish it. It took six
months of dedicated effort, but the individual's skin complexion returned
to absolute normal. Furthermore, the individual stopped taking the
supplement program, resumed silver use, and his skin began to change once
again. Whereby he resumed the supplementation, and the skin returned to
normal. Apparently, the silver build-up in the body when it reaches high
enough levels to deposit visible silver in the skin is quite extensive.
The process of removal is slow, but effective. Furthermore, the individual
actually had spirochetes in the eyes; the only thing that remains is
slight scar tissue, and the individual, who was losing the use of the
eyes, can see perfectly fine. The list of symptoms with such an event is
extensive. The individual was near liver failure, and within three months
of initial silver use, was all but completely restored to full health.
Fevers spiking 3-5 times daily, a chronic and severe lung infection,
inflammation of the liver, shut down of the body's elimination systems,
loss of reduction of cognitive ability, extreme and disabling fatigue are
among the symptoms that fell to the power of this...Silver Chloride.
Obviously silver chloride as a product is not equal to the superior
isolated silver products. However, the question remains: Would consuming
isolated silver have delivered enough actual silver content to the body to
be effective in this case?
A Cure for Argyria: The Formula
3 Vitamin E 1000 mg 100% Natural d-alpha Tocopheryl
1 Selenium 100mcg (yeast free)
2 vegetarian Vitamin C 1000 mg
1 teaspoon MSM organic
1 super potency Vitamin B 100,
1 teaspoon of Kelp powder:
Taken every morning with two 16oz glasses of water, with close to a total
of 3/4 of a gallon drinking water a day."
Just as an FYI, the following are from Appendix 9 of âSpacecraft Water
Exposure Guidelines for Selected Contaminants: Volume 1â. Free download
of book available at http://www.nap.edu/catalog.php?record_id=10942). It
provides an extensive review of silver toxicity and was performed for
NASA's use of silver in space vehicle water treatment.
âEven though silver salts are not metabolized in the typical sense, silver
salts that are transformed are reduced to metallic silver. It was suggested
(ATSDR 1990) that the deposition of silver in tissues is the result of
precipitation
of insoluble silver chlorides and silver phosphates and that those
silver salts are transformed to silver sulfides by forming complexes with
amino or carboxyl groups in proteins or are reduced to metallic silver by
reduction with ascorbic acid (Danscher 1981). Buckley et al. (1965) identified
silver particles deposited in the dermis of a woman with argyria as
silver sulfide. Similarly, Berry and Galle (1982) reported that deposits of
silver in the internal organs of rats were identified as silver sulfide.
Silver
seems to interact with other metal salts, especially with selenium in the diet
(Berry and Galle 1982, as cited in ATSDR 1990)."
"Factors That Influence Silver Toxicity
Diplock et al. (1967) reported that vitamin E and selenium in the diet
could significantly influence the toxicity of silver. When weanling Norwegian
hooded rats fed a basal vitamin-E deficient diet were provided drinking
water containing silver at 970 mg/L (as silver acetate), all rats developed
liver necrosis within 2-4 wk and died. In another group, when selenium
was added at 1 ppm to the vitamin-E deficient diet, and the drinking water
contained silver acetate, only four of nine rats died. In another group that
was fed a diet containing vitamin E and was sacrificed after 50 d of silver
exposure, no liver necrosis was found. Bunyan et al. (1968) reported similar
observations in rats exposed to silver at 650 mg/L (as silver acetate) in
drinking water. Liver necrosis was seen when the dietary selenium was
reduced. Necrosis was induced at much lower doses of silver (80 mg/L).
Vitamin E appeared to reverse that effect. Also, Grasso et al. (1969) reported
that when silver (silver acetate) was fed either in the diet (at
130-1,000 ppm, or 4-33 mg/kg/d) or in drinking water (97.5 mg/kg/d) to
vitamin-E deficient rats, fatal necrosis was noted. Alexander and Aaseth
(1981) reported that depletion of liver GSH by diethyl maleate decreased
biliary excretion of silver into the bile. Selenite also inhibited the biliary
excretion of silver and increased its retention in the tissues. It was
suggested
that selenite formed an insoluble complex with silver that retarded
biliary excretion. It is not clear if that is in any way related to the
effect of
selenium-containing diets in reducing the GSH peroxidase (see Wagner et
al. 1975, described above)."
- Steve N
________________________________
From: Silver Smith [mailto:[email protected]]
Sent: Tuesday, June 02, 2009 7:21 PM
To: [email protected]
Subject: CS>Getting Rid of "Blue Moons"
I recently had a friend point out that I am developing "Blue Moons" on my
fingernails. Can anybody recommend a remedy? How long should it take to
get rid of them?
I think they started when I was experimenting with CS as a deodorant and
mixed the CS with some of a "mineral stone" deodorant. The combo has
worked great for a year or so as a deodorant...NO odor! Kinda bummed
with the Blue Moons...it creates a bit of a problem when I am recommending
CS to someone and they are concerned with "turning blue".
Thanks,
SS
N'[Þ¬¸¬¶+^«^uú+ºgèجrë,x¢YhÖ¥J)oz²'²ÚîrبÇè®éì¹»®&â«z,µçZ¶m§ÿì[Þ®X¬¶àNh²Öv·¬³*.®g¬±¨¶"÷«+-zÉ"(]ë,9÷Ó¢ë,j¬¶"÷«¡÷í¢+-zÉ"(8^J)oz²â²Ö§tçßNbp¸¬µªÜ+Þ±ªÞrêëz{eÉÚ0¸¬¶f¢Ö¢êÌGzú.®g^¾«zÉ"(
--
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